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Anti-Insulin -Like Growth Factor II, Human Antibody
品牌:Abmgood
货号:Y103559
规格:
货期:

Anti-Insulin -Like Growth Factor II, Human Antibody

商品详情 参考文献 相关资料
Overview Print Version
Product Name Anti-Insulin -Like Growth Factor II, Human Antibody
Description W2H1
Subtype monoclonal
Application IHC, ELISA, RIA, Other
Raised In mouse
Specificity Human. Reactivity with other species has not been tested. This antibody recognizes IGF-II, crossreactivity with IGF-I <0.01% and insulin <0.01%
Immunogen Human anti-globulin conjugated to IGF-II
Relevance The IGF2 gene encodes a member of the insulin family of polypeptide growth factors that is involved in development and growth. It is expressed only from the paternally inherited allele and is an imprinted gene which is a candidate gene for eating disorders. There is a read-through, INS-IGF2, which aligns to this gene at the 3' region and to the upstream INS gene at the 5' region. Two alternatively spliced transcript variants encoding the same protein have been found for this gene. Insulin Like Growth Factor-2 (IGF-2) is a polypeptide growth factor, which stimulates the proliferation of a wide range of cell types
Guarantee abm guarantees that all our Anti-Insulin -Like Growth Factor II, Human Antibody will perform as described on this product webpage, if this is not the case we will provide you with a one-time replacement at no extra cost. Documentation and explanation of experiment conducted will be required when submitting a claim for replacement.
Properties
Formulation Liquid
Quantity 100 μg
Clone Monoclonal Antibody
Storage This product is stable for several weeks at 4°C as an undiluted liquid. Dilute only prior to immediate use. For extended storage, aliquot contents and freeze at -20°C or below. Avoid cycles of freezing and thawing. Expiration date is one (1) year from date of receipt.
Storage Buffer PBS with 10 mM sodium azide.
Purity Affinity purified
Isotype Human anti-globulin conjugated to IGF-II
Species Reactivity human
Specific References
Y103559 has not yet been referenced specifically in any publications.
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